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Aoxing Pharmaceutical Company (AMEX:AXN) is a US incorporated specialty pharmaceutical company with its operations in China, specializing in research, development, manufacturing and distribution of a variety of narcotics and pain-management products. Headquartered in Shijiazhuang City, outside Beijing, Aoxing has the largest and most advanced manufacturing facility in China for highly regulated narcotic medicines. Its facility is one of the few GMP facilities licensed for the manufacture of narcotic medicines by the China State Food and Drug Administration (SFDA). It has a joint venture collaboration with Johnson Matthey Plc to produce and market narcotics and neurological drugs in China. It also has strategic alliance partnership with QRxPharma, Phoenix PharmaLabs, Inc and American Oriental Bioengineering, Inc.

Even if we obtain FDA approval of zilucoplan or any of our other product candidates, we or our partners may never obtain approval or commercialize our products outside of the U.S.

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Vivier, E., Tomasello, E., Baratin, M., Walzer, T. & Ugolini, S. Functions of natural killer cells. Nat. Immunol. 9, 503–510 (2008).

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I saw in an earlier thread that you showed interest for GHK, could you elaborate a bit? have you tried it?

(a and b) Tumor cells induce ADPR production but cADPR in Cd38+/+ NK cells. Levels of ADPR and cADPR were determined after treatment of NK cells with 30 μg PME prepared from B16F10 tumor cells for 40 s. Data are mean ± SEM of three independent experiments. *P < 0.001; #P < 0.05. ns, not significant. (c–e) Cibacron blue 3GA (CB) inhibits of tumor-stimulated ADPR induction (c), sustained Ca2+ signal (d), cytotoxicity (e). CB (100 μM), ara-2′-F-NAD (200 nM), bafilomycin A1 (200 nM) or GPN (50 μM) was preincubated for 30 min. Data shown in c–e are representative of three independent experiments. *P < 0.001 vs basal; #P < 0.05.

To test the contribution of store-operated Ca2+ entry (SOCE) to the tumor cell–induced sustained Ca2+ signal, we used Baf A1 to deplete Ca2+ from acidic organelles, which are the Ca2+ stores responsible for tumor cell-mediated Ca2+ signals stimulated by ADPR (Fig. 4c and d). Depletion of Ca2+ stores by Baf A1 induced Ca2+ entry after Ca2+ was added to the bath perfusion solution, as described earlier36 (Fig. 5d). This SOCE was completely blocked by SK96365, a SOCE blocker, but not by ACA, a specific TRPM2 channel blocker. These findings indicate that the sustained Ca2+ signal is attributable to SOCE following Ca2+ mobilization by ADPR, and that TRPM2 is not involved in ADPR-mediated SOCE.

        As of March 1, 2019, we had 10 stockholders of record. The number of holders is based upon the actual number of holders registered in our records at such date and excludes holders in "street name" or persons, partnerships, associations, corporations, or other entities identified in security positions listings maintained by depository trust companies.

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Generally, under the Companies Law, the decision to distribute dividends and the amount to be distributed is made by a company’s board of directors. The Articles provide that the Board may from time to time declare, and cause the Company to pay, such dividends as may appear to it to be justified by the profits of the Company and that the Board has the authority to determine the time for payment of such dividends and the record date for determining the shareholders entitled to receive such dividends, provided the date is not before the date of the resolution to distribute the dividend. Declaration of dividends does not require shareholder approval.

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